Mouse study sheds light on maternal infection, autism link
A new study points to a possible link between inflammation in the womb, brain overgrowth and the behavioral impairments seen in autism.
From parental age to infection during pregnancy, environmental elements can influence autism risk.
A new study points to a possible link between inflammation in the womb, brain overgrowth and the behavioral impairments seen in autism.
Rats exposed in utero to the epilepsy drug valproic acid, a risk factor for autism, do not develop autism-like behaviors if they are reared in a stimulating environment. Researchers presented the unpublished findings yesterday at the 2014 Society for Neuroscience annual meeting in Washington, D.C.
Loss of MeCP2, the Rett syndrome gene, depletes immune cells throughout the bodies of mice, researchers reported yesterday at the 2014 Society for Neuroscience annual meeting in Washington, D.C.
Mice born to older males with mutations in PAX6 — a gene involved in brain development — vocalize less than those with younger dads. The unpublished findings, presented today at the 2014 Society for Neuroscience annual meeting in Washington, D.C., suggest how genes and paternal age can work together to trigger symptoms.
Soft touch and physical closeness to other people wire the social brain right from the earliest days after birth, and problems in the response to touch may play a fundamental role in autism. This picture emerges from unpublished results presented by several teams at the 2014 Society for Neuroscience annual meeting in Washington, D.C.
Mice born via cesarean section show subtle social deficits and increased immune and stress responses, suggesting a link between microbe exposure during birth and behavior. Researchers presented the unpublished work today at the 2014 Society for Neuroscience annual meeting in Washington, D.C.
Infection with group B streptococcus bacteria in pregnant rats triggers brain abnormalities and autism-like behaviors in their pups — especially in males. Researchers presented the unpublished results today at the 2014 Society for Neuroscience annual meeting in Washington, D.C.
Valproic acid, an epilepsy drug that increases the risk of autism, may alter neural circuits by opening up chromatin, a tightly packaged form of DNA in the nucleus. Researchers presented the unpublished data today at the 2014 Society for Neuroscience annual meeting in Washington, D.C.
Some cases of autism may result from glitches in immune cells in the blood: This provocative idea stems from a series of unpublished mouse studies presented yesterday at the 2014 Society for Neuroscience annual meeting in Washington, D.C.
Monkeys exposed in utero to their mother’s immune response to a mock infection show inflammation in their brains four years later. Researchers presented the unpublished work today at the 2014 Society for Neuroscience annual meeting in Washington, D.C.